Research areas
Understanding our focus

Mechanisms of estrogen modulation of neural circuitry
The neurosteroid estrogen is fast emerging as a key regulator of synaptic plasticity, but how estrogen achieves this is still unclear.
So far the Srivastava lab has:
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Identified key signalling pathways involved in estrogenic-modulation of synaptic plasticity.
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Identified a novel form of spine plasticity, whereby estrogen “prime” neurons to respond to subsequent synaptic-activity stimuli with greater efficacy. This is achieved by estrogen acutely modulating spine structure and functional plasticity in neural circuits.
We are now focused on dissecting the molecular mechanisms and signalling pathways that underlie this novel form of plasticity in vitro as well as in vivo.
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This will provide a mechanistic understanding of how this neurosteroid controls structural and functional plasticity and how this regulation may be relevant for its effects on cognition.
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Additionally, we are using neurons generated from patient specific induced pluripotent stem cells (iPSCs) to investigate the potential therapeutic value, and underlying mechanisms, of estrogen-based compounds in treatment of schizophrenia.
Recent work in the lab has focused on:
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Using human neural stem cells as an additional cellular model to further elucidate mechanisms that are critical for the development of neurodevelopmental disorders.
Utilizing this approach, as well as neurons differentiated from human induced pluripotent stem cells (hiPSC-neurons).
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We have investigated the biological function of ZNF804a, the first gene to be identified by GWAS studies as a risk factor for schizophrenias, bi-polar and more recently, autism spectrum disorders.
This work has identified that ZNF804a regulates early development of neuronal morphology, and moreover, is required for the maintenance of synapse number.
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We are also utilized patient specific hiPSC-neurons from individuals diagnosed with schizophrenia or autism spectrum disorders (ASDs), to investigate the mechanisms that contribute to the emergence of these disorders.
